Neus Prats

    <p>Neus Prats</p>
    Research Group: 
    Histopathology Facility
    Position: 
    Core Facility Manager
    Office: 
    EMS1C14
    Telephone: 
    +34 93 40 20546
    Email: 
    neus.prats@irbbarcelona.org

     

    View research group

    Selected group publications

    Identity Noise and Adipogenic Traits Characterize Dermal Fibroblast Aging.
    Salzer MC, Lafzi A, Berenguer-Llergo A, Youssif C, Castellanos A, Solanas G, Peixoto FO, Stephan-Otto Attolini C, Prats N, Aguilera M, Martín-Caballero J, Heyn H, Benitah SA.
    Identity Noise and Adipogenic Traits Characterize Dermal Fibroblast Aging. Cell, 6 (175), doi: 10.1016/j.cell.2018.10.012-1575-1590 (2019)
    Defects in efferent duct multiciliogenesis underlie male infertility in GEMC1-, MCIDAS- or CCNO-deficient mice.
    Terré B, Lewis M, Gil-Gómez G, Han Z, Lu H, Aguilera M, Prats N, Roy S, Zhao H, Stracker TH.
    Defects in efferent duct multiciliogenesis underlie male infertility in GEMC1-, MCIDAS- or CCNO-deficient mice. Development, 8 (146), doi: 10.1242/dev.162628 (2019)
    BMAL1-Driven Tissue Clocks Respond Independently to Light to Maintain Homeostasis.
    Welz PS, Zinna VM, Symeonidi A, Koronowski KB, Kinouchi K, Smith JG, Guillén IM, Castellanos A, Furrow S, Aragón F, Crainiciuc G, Prats N, Caballero JM, Hidalgo A, Sassone-Corsi P, Benitah SA.
    BMAL1-Driven Tissue Clocks Respond Independently to Light to Maintain Homeostasis. Cell, 6 (177), doi: 10.1016/j.cell.2019.05.009-1436-1447 (2019)
    Loss of G9a preserves mutation patterns but increases chromatin accessibility, genomic instability and aggressiveness in skin tumours.
    Avgustinova A, Symeonidi A, Castellanos A, Urdiroz-Urricelqui U, Solé-Boldo L, Martín M, Pérez-Rodríguez I, Prats N, Lehner B, Supek F, Benitah SA.
    Loss of G9a preserves mutation patterns but increases chromatin accessibility, genomic instability and aggressiveness in skin tumours. Nat Cell Biol., 12 (20), doi: 10.1038/s41556-018-0233-x-1400-1409 (2018)
    Targeting metastasis-initiating cells through the fatty acid receptor CD36.
    Pascual G, Avgustinova A, Mejetta S, Martín M, Castellanos A, Attolini CS, Berenguer A, Prats N, Toll A, Hueto JA, Bescós C, Di Croce L, Benitah SA.
    Targeting metastasis-initiating cells through the fatty acid receptor CD36. Nature, 7635 (541), doi: 10.1038/nature20791.-41-45 (2017)
    A genome editing approach to study cancer stem cells in human tumors.
    Cortina C, Turon G, Stork D, Hernando-Momblona X, Sevillano M, Aguilera M, Tosi S, Merlos-Suárez A, Stephan-Otto Attolini C, Sancho E, Batlle E.
    A genome editing approach to study cancer stem cells in human tumors. EMBO Mol Med., 7 (9), doi: 10.15252/emmm.201707550.-869-879 (2017)
    Loss of Dnmt3a and Dnmt3b does not affect epidermal homeostasis but promotes squamous transformation through PPAR-γ.
    Rinaldi L, Avgustinova A, Martín M, Datta D, Solanas G, Prats N, Benitah SA.
    Loss of Dnmt3a and Dnmt3b does not affect epidermal homeostasis but promotes squamous transformation through PPAR-γ. Elife, doi: 10.7554/eLife.21697. (2016)
    Full publication list